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APOL1 Risk Variants Associate With the Prevalence of Stroke in African American Current and Past Smokers.
Rakic JM, Pullinger CR, Van Blarigan EL, Movsesyan I, Stock EO, Malloy MJ, Kane JP. Rakic JM, et al. Among authors: pullinger cr. J Am Heart Assoc. 2023 Dec 19;12(24):e030796. doi: 10.1161/JAHA.123.030796. Epub 2023 Dec 12. J Am Heart Assoc. 2023. PMID: 38084718 Free PMC article.
The association between smoking and stroke differed by APOL1 genotype (P(interaction term)=0.014). Among carriers, ever versus never smokers had odds ratio (OR) 2.46 (95% CI, 1.08-5.59) for stroke (P=0.034); OR 2.00 (95% CI, 0.81-4.96) among carriers of 1 risk allele, and …
The association between smoking and stroke differed by APOL1 genotype (P(interaction term)=0.014). Among carriers, ever versus never …
APOL1 Risk Variants Associate with the Prevalence of Stroke in African American Current and Past Smokers.
Rakic JM, Pullinger CR, Van Blarigan EL, Movsesyan I, Stock EO, Malloy MJ, Kane JP. Rakic JM, et al. Among authors: pullinger cr. medRxiv [Preprint]. 2023 Apr 29:2023.04.28.23289292. doi: 10.1101/2023.04.28.23289292. medRxiv. 2023. PMID: 37162992 Free PMC article. Updated. Preprint.
The association of smoking history and stroke differed by APOL1 genotype status in the unstratified model (P(interaction term)=0.016). Among carriers of risk variants, ever smokers had odds ratio (OR) =2.88 for stroke compared to never smokers (P=0. 0.038). ...
The association of smoking history and stroke differed by APOL1 genotype status in the unstratified model (P(interaction term)=0.016) …
Epigenetic regulation and measurement of epigenetic changes.
Stephens KE, Miaskowski CA, Levine JD, Pullinger CR, Aouizerat BE. Stephens KE, et al. Among authors: pullinger cr. Biol Res Nurs. 2013 Oct;15(4):373-81. doi: 10.1177/1099800412444785. Epub 2012 Jun 3. Biol Res Nurs. 2013. PMID: 22661641 Free PMC article. Review.
These changes may additively or synergistically influence gene expression and result in long-term molecular and functional consequences independent of the DNA sequence that may ultimately define an individual's phenotype. ...
These changes may additively or synergistically influence gene expression and result in long-term molecular and functional consequenc …
Truncated variants of apolipoprotein B cause hypobetalipoproteinaemia.
Collins DR, Knott TJ, Pease RJ, Powell LM, Wallis SC, Robertson S, Pullinger CR, Milne RW, Marcel YL, Humphries SE, et al. Collins DR, et al. Among authors: pullinger cr. Nucleic Acids Res. 1988 Sep 12;16(17):8361-75. doi: 10.1093/nar/16.17.8361. Nucleic Acids Res. 1988. PMID: 2843815 Free PMC article.
Here we describe mutations of the apo-B gene that cause two different truncated variants of apo-B in unrelated individuals with hypobetalipoproteinaemia. One variant, apo-B(His1795----Met-Trp-Leu-Val-Thr-Term) is predicted to be 1799 amino acids long and arises from deleti …
Here we describe mutations of the apo-B gene that cause two different truncated variants of apo-B in unrelated individuals with hypobetalipo …
Diurnal changes in the rate of cholesterogenesis in hepatocytes from fed and starved rats: effects of precursors and pancreatic hormones in vitro.
Björnsson OG, Pullinger CR, Gibbons GF. Björnsson OG, et al. Among authors: pullinger cr. Arch Biochem Biophys. 1985 Apr;238(1):135-45. doi: 10.1016/0003-9861(85)90149-3. Arch Biochem Biophys. 1985. PMID: 3885855
In hepatocytes prepared from animals starved for a longer period (43 h) the decrease in the activity of HMG-CoA reductase was much less than that in the rate of sterol synthesis. In contrast to hepatocytes from fed or short-term-starved animals, the rate of sterol synthesi …
In hepatocytes prepared from animals starved for a longer period (43 h) the decrease in the activity of HMG-CoA reductase was much less than …