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Posttreatment reactivation of tuberculosis in mice caused by Mycobacterium tuberculosis disrupted in mce1R.
Cheigh CI, Senaratne R, Uchida Y, Casali N, Kendall LV, Riley LW. Cheigh CI, et al. Among authors: casali n. J Infect Dis. 2010 Sep 1;202(5):752-9. doi: 10.1086/655224. J Infect Dis. 2010. PMID: 20629532 Free PMC article.
The constitutive expression of the mce1 genes in the mce1R mutant may allow this mutant to maintain its lipid metabolism, enabling it to survive long-term and proliferate inside granulomas....
The constitutive expression of the mce1 genes in the mce1R mutant may allow this mutant to maintain its lipid metabolism, enabling it to sur …
Accelerated immunopathological response of mice infected with Mycobacterium tuberculosis disrupted in the mce1 operon negative transcriptional regulator.
Uchida Y, Casali N, White A, Morici L, Kendall LV, Riley LW. Uchida Y, et al. Among authors: casali n. Cell Microbiol. 2007 May;9(5):1275-83. doi: 10.1111/j.1462-5822.2006.00870.x. Epub 2007 Jan 11. Cell Microbiol. 2007. PMID: 17223927
These observations suggest that the early deregulated expression of the mce1 operon products determines later granulomatous tissue response. mce1 operon may homeostatically regulate the cell wall architecture in vivo that elicits a steady-state granuloma tissue response permittin …
These observations suggest that the early deregulated expression of the mce1 operon products determines later granulomatous tissue response. …