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Dual antiplatelet therapy prolongation in high-risk patients with prior myocardial infarction: insights from the post-PCI registry.
Ferlini M, Rossini R, Musumeci G, Cornara S, Somaschini A, Grieco N, Marino M, Calchera I, Cardile A, Colombo P, Martinoni A, Ielasi A, Castiglioni B, Lettieri C, Tarantini G, Oltrona Visconti L. Ferlini M, et al. Among authors: cardile a. J Cardiovasc Med (Hagerstown). 2020 Aug;21(8):603-609. doi: 10.2459/JCM.0000000000000988. J Cardiovasc Med (Hagerstown). 2020. PMID: 32520857
BACKGROUND: Patients surviving a myocardial infarction (MI) are at a heightened risk for recurrent ischemic events that can be reduced with the long-term addition of a second antithrombotic drug to aspirin. However, data about real prescription of this therapy are lacking …
BACKGROUND: Patients surviving a myocardial infarction (MI) are at a heightened risk for recurrent ischemic events that can be reduced with …
Mitochondrial biogenesis by NO yields functionally active mitochondria in mammals.
Nisoli E, Falcone S, Tonello C, Cozzi V, Palomba L, Fiorani M, Pisconti A, Brunelli S, Cardile A, Francolini M, Cantoni O, Carruba MO, Moncada S, Clementi E. Nisoli E, et al. Among authors: cardile a. Proc Natl Acad Sci U S A. 2004 Nov 23;101(47):16507-12. doi: 10.1073/pnas.0405432101. Epub 2004 Nov 15. Proc Natl Acad Sci U S A. 2004. PMID: 15545607 Free PMC article.
We recently found that long-term exposure to nitric oxide (NO) triggers mitochondrial biogenesis in mammalian cells and tissues by activation of guanylate cyclase and generation of cGMP. ...
We recently found that long-term exposure to nitric oxide (NO) triggers mitochondrial biogenesis in mammalian cells and tissues by ac …